Assignment: Pathophysiological Disease Process
Assignment: Pathophysiological Disease Process
Assignment: Pathophysiological Disease Process
Pathophysiological disease process assignment: The purpose of the case study is to have you expand upon the pathophysiological disease process by searching for evidence-based practice treatment and advanced practice nursing role implications related to the disease.
Case Study Topic on Pathophysiological disease process assignment:
Select a type of cancer (breast, lung, prostate, or colon cancer, melanoma, or any type of leukemia) and discuss the relationship of the disease process to the immune system.
according to Assignment: Pathophysiological Disease Process , Identify current evidence-based treatment modalities for the selected cancer and discuss how the treatment impacts the disease process.
Conduct an evidence-based literature search to identify the most recent standards of care/treatment modalities from peer-reviewed articles and professional association guidelines (www.guideline.gov). These articles and guidelines can be referenced, but not directly copied into the clinical case presentation. Cite a minimum of three resources.
Assignment: Pathophysiological Disease Process
Include the following in your clinical case presentation on Pathophysiological disease process assignment:
A discussion of the pathophysiology of the disease, including signs and symptoms
An explanation of diagnostic testing and rationales for each
A review of different evidence-based treatment modalities for the disorder obtained from guideline.gov or a professional organization such as thyroid (American Thyroid Association), OB-GYN (ACOG), urology (AUA), etc.
Next, address the following questions:
How does the information, in this case, inform the practice of a master’s prepared nurse?
How should the master’s prepared nurse use this information to design a patient education session for someone with this condition?
What was the most important information presented in this case?
What was the most confusing or challenging information presented in this case?
Discuss a patient safety issue that can be addressed for a patient with the condition presented in this case.
The use of medical terminology and appropriate graduate level writing is expected.
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Pathophysiological disease process assignment requirements
Your paper should be 3–4 pages (excluding cover page and reference page).
Your resources must include research articles as well as reference to non-research evidence-based guidelines. ( No more than 3 years published)
Use APA format to style your paper and to cite your sources. Your source(s) should be integrated into the paragraphs. Use internal citations pointing to evidence in the literature and supporting your ideas. You will need to include a reference page listing those sources. Cite a minimum of three resources.
Review the rubric for more information on how your assignment will be graded.
https://www.google.com/search?q=google&oq=google&aqs=chrome..69i57j46i67i131i199i433i465j69i59j0i67i433j0i67i131i433j69i60l3.3914j0j7&sourceid=chrome&ie=UTF-8
Use APA format to style your paper and to cite your sources. Your source(s) should be integrated into the paragraphs. Use internal citations pointing to evidence in the literature and supporting your ideas. You will need to include a reference page listing those sources.
Assignment: Pathophysiological Disease Process
The pathophysiology of Parkinson’s disease is defined by the death of dopaminergic neurons as a result of alterations in biological activity in the brain (PD). There are various potential causes for neuronal death in Parkinson’s disease, although not all of them have been thoroughly investigated. Protein aggregation in Lewy bodies, disruption of autophagy, alterations in cell metabolism or mitochondrial function, neuroinflammation, and blood-brain barrier (BBB) collapse resulting in vascular leakiness are five postulated primary pathways for neuronal death in Parkinson’s Disease. [18]
Heart failure is caused by a decrease in the effectiveness of the heart muscle as a result of damage or overloading. As a result, it can be caused by a variety of conditions, including myocardial infarction (in which the heart muscle is starved of oxygen and dies), hypertension (in which the force of contraction required to pump blood is increased), and amyloidosis (in which the heart muscle is starved of oxygen and dies) (in which misfolded proteins are deposited in the heart muscle, causing it to stiffen). These increased workloads will cause alterations in the heart over time.
Multiple sclerosis is an inflammatory demyelinating disease of the central nervous system in which activated immune cells infiltrate the CNS and produce inflammation, neurodegeneration, and tissue destruction. The underlying disease that causes this behavior is unknown at this time. Current research in neuropathology, neuroimmunology, neurobiology, and neuroimaging, as well as clinical neurology, support the idea that MS represents a spectrum of diseases rather than a single disease. [19]
Hypertension has a mechanism that is similar to that of a chronic disease marked by high blood pressure. Hypertension can be classed as either essential (also known as primary or idiopathic) or secondary (also known as secondary hypertension). Essential hypertension accounts for 90–95 percent of all hypertension. [20] [21] [22] [23]
The pathophysiology of HIV/AIDS involves the virus replicating inside and killing T helper cells, which are necessary for practically all adaptive immune responses, after infection. After an early period of influenza-like sickness, the virus becomes latent and asymptomatic. The HIV host has progressed to AIDS when the CD4 lymphocyte count falls below 200 cells/ml of blood, a state marked by a lack of cell-mediated immunity and increased vulnerability to opportunistic infections and some types of malignancies.
The venom’s action causes the pathophysiology of spider bites. When a spider injects venom into the skin, it is known as spider envenomation. A dry bite is one in which the spider bite does not inject venom, and the amount of venom injected varies depending on the kind of spider and the conditions of the encounter. Humans are not at risk from mechanical injuries caused by spider bites.
according to Assignment: Pathophysiological Disease Process , Obesity pathophysiology includes a variety of pathophysiological pathways that play a role in its development and maintenance.
[25] Until J. M. Friedman’s laboratory found the leptin gene in 1994, this field of research was almost unexplored. [26] Leptin was thought to be a satiety factor by these researchers. Mutations in the leptin gene resulted in the obese phenotype in the ob/ob animal, indicating the possibility of leptin therapy for human obesity. However, J. F. Caro’s group soon discovered that there were no mutations in the leptin gene in obese people. Leptin expression was raised on the other hand, suggesting the possibility of Leptin resistance in human obesity. [27]
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